Effect of rapamycin on cytokine profile in kidney transplant recipients treated with triple drug therapy

L. E. Morales-Buenrostro, L. Llorente, Y. Richaud-Patin, C. De Leo, E. Soto-Vega, A. Diáz-Alderete, L. G. Vázquez-Lavista, S. Coto, R. Correa-Rotter, J. Alberú

Research output: Contribution to journalArticlepeer-review

3 Scopus citations

Abstract

The aim of this study was to explore differences in the cytokine profile among de novo kidney transplant recipients treated with either Rapamycin (Rapa) + cyclosporine (CsA) + prednisone (P) or CsA + azathioprine (Aza) + P. Among the 13 adult kidney transplant recipients studied, seven received Rapa + CsA + P while the remaining six received CsA + Aza + P with their living donors serving as controls (n = 13). Spontaneous production of IL-2, IFNγ, IL-10, and TGF-β were measured by ELISA in supernatants from 24-hour cultured unstimulated peripheral blood mononuclear cell (PBMC) at time zero (the day before the transplant), and at 3 and 6 months posttransplant. Cytokines were also measured 1 month after CsA withdrawal in the Rapa + CsA + P group. From time zero to the end of the study, IL-2, IFNγ, and IL-10 were present at low or undetectable levels in all three groups. TGF-β tended to increase in supernatants from patients under Rapa + CsA + P at 6 months posttransplant and at 1 month after CsA withdrawal without correlation to Rapa blood levels. TGF-β remained stable throughout the study period for patients included in the CsA + Aza + P group. There was no difference in this cytokine level between these study groups at any given time. This study showed no differences in the spontaneous cytokine profiles evaluated in patients treated with both therapeutic schemes.

Original languageEnglish
Pages (from-to)1661-1663
Number of pages3
JournalTransplantation Proceedings
Volume36
Issue number6
DOIs
StatePublished - 1 Jul 2004
Externally publishedYes

Fingerprint

Dive into the research topics of 'Effect of rapamycin on cytokine profile in kidney transplant recipients treated with triple drug therapy'. Together they form a unique fingerprint.

Cite this